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Androgenetic alopecia, or baldness, can result from the use of anabolic-androgenic steroids (AAS).

At around age 50, more than half of all men develop this type of alopecia. In this instance, hair loss exhibits clear signs rather than developing spontaneously.

• Baldness is distributed differently between men and women.

Here, there is a definite relationship between the location of androgen receptors on the scalp and the area of hair loss. In men, hair loss in the frontal and parietal regions results in the development of a bald head; in women, hair loss occurs on both sides of the central part and the vertex, but no bald spot forms.

• The active hair growth phase lasts for a shorter amount of time.

The main stage is the resting one when the hair stops growing and starts to fall out. This can be done simply by washing and combing the hair.

Hair follicles stop functioning normally as the number of them declines, eventually filling with connective tissue and eventually dying.

Both androgenic factors and genetic predisposition affect those who are susceptible to androgenetic alopecia. They develop more dihydrotestosterone in the scalp and have more androgen receptors than people who aren’t prone to baldness.

WHAT CAUSES ALOPECIA?

Alopecia is primarily brought on by dihydrotestosterone (DHT). It is created from testosterone by an enzyme that is constantly present in the hair follicle. The blood vessels feeding the follicles spasm when DHT is present, and in place of thick, full hair, they start to grow thin, short, slightly dyed hair.

Since these vessels are unable to cover the scalp with hair, the scalp is visible through them, giving the appearance of visible alopecia. Hair follicles eventually stop producing hair and slowly disappear as a result of the constant lack of nutrition. Even in the absence of the necessary level of testosterone, the same principle holds for all AAS.

It is still unclear how much baldness is genetic. In the past, it was thought that the mother received the inheritance from the grandfather, but new evidence points to the father as the source of the baldness predisposition. It demonstrates that both the onset of alopecia and its severity is caused by a gene complex rather than a single gene.

WHAT MEDICAL PROCEDURE CAN TREAT ALOPECIA?

Dutasteride and Finasteride are used to treat androgenetic baldness because they inhibit the enzyme that turns testosterone into dihydrotestosterone.

Oestrogens and anti-androgenic medications are recommended for the treatment of women. The first step in treatment is to lessen the impact of androgens, which helps to significantly reduce the severity of alopecia.

Because of this, athletes who use steroids to gain muscle choose them carefully to reduce unneeded androgenic stimulation. This is accomplished by using the right steroid substances, such as oxandrolone (Anavar), nandrolone (Deca), and methenolone enanthate (Primobolan), in the right dosages. To reduce the production of dihydrotestosterone in the scalp as an alternative, injectable testosterone preparations in combination with finasteride can be used.

WILL EVERYONE WHO USES STEROIDS DEVELOP ALOPECIA?

The relationship between baldness and AAS reception has not yet been investigated. Nevertheless, using AAS causes more hair loss in those who already have androgenetic alopecia. If hair loss was previously unproblematic, then alopecia can only be noted with the use of these medications as a side effect, manifesting itself not in everyone even after years of taking AAS.

These facts support the idea that androgens only cause baldness in situations where there is a genetic predisposition to it. It cannot be assumed that AAS will inevitably result in baldness if there is no such tendency, even in the absence of necessary conditions.

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